ECG Workshop Nezar Amir Case one A 61-year-old man with a history - - PowerPoint PPT Presentation

ecg workshop
SMART_READER_LITE
LIVE PREVIEW

ECG Workshop Nezar Amir Case one A 61-year-old man with a history - - PowerPoint PPT Presentation

ECG Workshop Nezar Amir Case one A 61-year-old man with a history of hypertension and congestive heart failure presents to the emergency department with shortness of breath after eating breakfast. All of the following statements about his ECG


slide-1
SLIDE 1

ECG Workshop

Nezar Amir

slide-2
SLIDE 2

Case one

A 61-year-old man with a history of hypertension and congestive heart failure presents to the emergency department with shortness of breath after eating breakfast. All of the following statements about his ECG are correct EXCEPT: a) The QRS axis is normal b) The rhythm is sinus tachycardia c) The PR interval is within normal limits d) There is a complete left bundle branch block e) The voltage in the chest leads meets criteria for left ventricular hypertrophy

slide-3
SLIDE 3

a) The QRS axis is normal b) The rhythm is sinus tachycardia c) The PR interval is within normal limits d) There is a complete left bundle branch block e) The voltage in the chest leads meets criteria for left ventricular hypertrophy

slide-4
SLIDE 4

The criteria for complete LBBB include:

  • 1. QRS duration > 0.12 second
  • 2. A wide deep QS complex in V1
  • 3. A wide tall R wave in V6

The correct answer is d) There is no left bundle branch block (LBBB)

slide-5
SLIDE 5

LVH

1.Prominent voltage in the chest leads and selected limb lead 2.Widened QRS 3.T wave inversions in leads with tall R waves 4. Left axis deviation

  • 5. Voltage criteria for left ventricular hypertrophy (LVH) should be used

with caution. Commonly used voltage criteria include

  • ne
  • r

1. SV1 + RV5

  • r

V6 > 35 mm (3.5 mV) 2. RaVL > 11 mm (1.1 mV) 3. For men: SV3 + RaVL > 28 mm (2.8 mV) 4. For women: SV3 + RaVL > 20 mm (2.0 mV)

slide-6
SLIDE 6

Case two A 26-year-old woman comes to the emergency department complaining of increased shortness of breath. Which one of the following statements is true concerning her admission ECG? a) The PR interval is prolonged b) The QRS axis is normal c) There is normal R wave progression d) There is a complete right bundle branch block e) There is evidence of right ventricular hypertrophy

slide-7
SLIDE 7

a) The PR interval is prolonged b) The QRS axis is normal c) There is normal R wave progression d) There is a complete right bundle branch block e) There is evidence of right ventricular hypertrophy

slide-8
SLIDE 8

Criteria for RVH

  • 1. RAD- axis is perpendicular to AVF~180
  • 2. qR in V1
  • 3. ST-T changes in V1-V4 in keeping with RV strain pattern

Correct Answer is e

slide-9
SLIDE 9

RV hypertrophy occurs over time in response to pressure or volume

  • verload in conditions such as;

1. Primary pulmonary hypertension 2. Chronic obstructive pulmonary disease (COPD) 3. Pulmonic stenosis 4. Atrial septal defect (ASD). This patient was diagnosed with PAH

slide-10
SLIDE 10

Case 3; The above ECG is from a 64 year old Caucasian male referred by the primary care physician to the cardiac outpatient clinic because of a very abnormal ECG. The patient is asymptomatic, without any sort of chest pain, dyspnea, palpitations, or previous syncope or dizzy spells. The BP is 130/80 mmHg and there are not murmurs on auscultation.

slide-11
SLIDE 11

What would you do?

1. Urgent hospital admission for coronary arteriography

  • 2. Urgent angiographic CT scan to exclude

pulmonary embolism

  • 3. Consider this ECG as a normal variant and

reassure the patient accordingly

  • 4. Nothing, this is a typical artifact originating

from a poor connection of the Wilson terminal to the ground 5.None of the above

slide-12
SLIDE 12

ECGs similar to this one can be seen in 1. Athletes of African or Afro-American origin without the phenotype of hypertrophic cardiomyopathy: our patient is Caucasian and is not an athlete, but a 64 year old male in whom his primary care physician

  • btained a routine ECG

2. Severe hypertensive heart disease: the blood pressure in this patient was normal 3. Valvular aortic stenosis: there were no heart murmurs on auscultation 4. Hypertrophic cardiomyopathy: the absence of murmurs should prompt us to consider a non-obstructive hypertrophic cardiomyopathy

slide-13
SLIDE 13

cMR

slide-14
SLIDE 14

Case 4; The above ECG is from a 53 year old male with a history of high blood pressure for the last couple of years. He is overweight and has mild hyperglycemia. He is referred by the primary care physician to the cardiac outpatient clinic because of a history of episodes of palpitations during the last 3 months, unrelated to exercise, of a very short duration, two or 3 times per month. On auscultation there is a 2/6 systolic murmur along the left sternal border and a wide splitting of the second heart sound.

slide-15
SLIDE 15
slide-16
SLIDE 16

What would you do first?

1.Chest X ray 2.2D ECHO 3.Holter recording 4.CT scan 5.Cardiac MRI

slide-17
SLIDE 17
slide-18
SLIDE 18
slide-19
SLIDE 19

Case 5: This ECG from an 18 year old male shows all of the following EXCEPT? a) Normal variant early repolarization pattern b) Physiologic sinus arrhythmia c) Normal AV conduction d) Left axis deviation e) Transition zone in lead V3

slide-20
SLIDE 20

a) Normal variant early repolarization pattern b) Physiologic sinus arrhythmia c) Normal AV conduction d) Left axis deviation e) Transition zone in lead V3

slide-21
SLIDE 21

d) Left axis deviation This ECG shows a normal variant that is commonly referred to as early repolarization pattern." There are ST elevations in leads V2-V6 and in some of the limb leads. Slight notching of the terminal QRS (V4) is often seen in conjunction with this pattern. The ST segment retains its normal upward concavity. The QRS axis here is normal (about +30 degrees). The QRS transition zone (R=S) is in lead V3, a normal finding. AV conduction is normal, indicated by the normal PR interval (about 0.14 sec.) The slight variation in heart rate is due to physiologic (respiratory) sinus arrhythmia.

slide-22
SLIDE 22

Case 6 This ECG from a 23 year-old female is most consistent with which diagnosis? a) Left atrial abnormality b) Anterior ischemia c) Normal variant T wave inversions V1-V2 d) Hypokalemia e) Left ventricular hypertrophy

slide-23
SLIDE 23

a) Left atrial abnormality b) Anterior ischemia c) Normal variant T wave inversions V1-V2 d) Hypokalemia e) Left ventricular hypertrophy

slide-24
SLIDE 24

ECG manifestations of acute myocardial ischemia

  • ST elevation

New ST elevation at the J-point in two contiguous leads with the cut-off points: ≥ 0.2 mV in men or ≥ 0.15 mV in women in leads V2-V3 and/or ≥ 0.1 mV in other leads.

  • ST depression and T-wave

changes

New horizontal or down-sloping ST depression > 0.05 mV in two contiguous leads: and/ or T inversion ≥ 0.1 mV in two contiguous leads with prominent R-wave or R/S ratio ≥ 1.

slide-25
SLIDE 25

ECG infarct

slide-26
SLIDE 26

Common causes of ST shift

slide-27
SLIDE 27

Infarct localisation

  • Left main artery occlusion:
  • diffuse ST-depression with ST

elevation in AVR

  • very high risk
  • Anterior wall:
  • ST elevation V1-V4. LAD. (often

tachycardia)

  • Inferior wall:
  • ST elevation II, III, AVF.
  • 80% RCA (elevation III>II; depression

> I or in AVL), or RCX ( in 20%). (often bradycardic due to sinus node

  • r AV node ischemia)
  • Right ventricle infarct:
  • ST elevation in V4R.
  • Posterior wall:
  • high R and ST-depression in V1-V3
  • Lateral wall:
  • ST elevation in lead I, AVL, V6.
  • LAD (D-branch)
slide-28
SLIDE 28

V4 right helps diagnose right ventricular involvement (in RCA occlusion)

slide-29
SLIDE 29

Acute inferior MI

slide-30
SLIDE 30

Old inferior MI: prominent Q waves in II, III & AVF

slide-31
SLIDE 31

Acute anterior-lateral infarct

slide-32
SLIDE 32

Acute antero-septal MI

slide-33
SLIDE 33

Recent (days old) anterior MI (after PCI)

slide-34
SLIDE 34

Old anterior-septal MI

slide-35
SLIDE 35

Acute posterior MI

more about this topic on ECGpedia...

slide-36
SLIDE 36

Notice the rather typical relative absence of ST deviation.

Acute RCX occlusion

slide-37
SLIDE 37

Old/recent posterior-lateral MI prominent R in V2 (a 'reciprocal Q wave')

slide-38
SLIDE 38

Acute inferior-posterior-lateral MI

slide-39
SLIDE 39

Acute inferior and right ventricular MI Elevation of V4R

slide-40
SLIDE 40

Left main disease Diffuse ST depression and elevation in AVR

slide-41
SLIDE 41

ST elevation in the absence of an aMI

Some other conditions that can cause ST elevation are:

  • Pericarditis/myocarditis.
  • Left ventricular hypertrophy (LVH)
  • Physiological/benign ST elevation
  • Cardiac aneurysm
  • Hyperkalemia
  • LBBB
  • HCM
slide-42
SLIDE 42

ST elevation in LBBB

slide-43
SLIDE 43

a) Complete right bundle branch block b) Complete left bundle branch block c) Wolff-Parkinson-White pre-excitation (right sided bypass tract) d) Left anterior fascicular block e) Left posterior fascicular block

slide-44
SLIDE 44

ST elevation in LVH

slide-45
SLIDE 45

ST elevation during high potassium levels

slide-46
SLIDE 46

Diffuse ST elevation in pericarditis

slide-47
SLIDE 47

Non-ST Elevation Infarction

Note the ST depression and T-wave inversion in leads V2-V6.

Here’s an ECG of an evolving non-ST elevation MI:

Question:

What area of the heart is infarcting?

Anterolateral

slide-48
SLIDE 48

Bundle Branch Blocks

slide-49
SLIDE 49

Bundle Branch Blocks

Turning our attention to bundle branch blocks… Remember normal impulse conduction is SA node  AV node  Bundle of His  Bundle Branches  Purkinje fibers

slide-50
SLIDE 50

Normal Impulse Conduction

Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers

slide-51
SLIDE 51

Bundle Branch Blocks

So, depolarization of the Bundle Branches and Purkinje fibers are seen as the QRS complex on the ECG. Therefore, a conduction block of the Bundle Branches would be reflected as a change in the QRS complex.

Right BBB

slide-52
SLIDE 52

Bundle Branch Blocks

With Bundle Branch Blocks you will see two changes on the ECG.

  • 1. QRS complex widens (> 0.12 sec).
  • 2. QRS morphology changes (varies depending on ECG lead, and if it is

a right vs. left bundle branch block).

slide-53
SLIDE 53

Bundle Branch Blocks

Why does the QRS complex widen? When the conduction pathway is blocked it will take longer for the electrical signal to pass throughout the ventricles.

slide-54
SLIDE 54

Right Bundle Branch Blocks

What QRS morphology is characteristic? V1 For RBBB the wide QRS complex assumes a unique, virtually diagnostic shape in those leads overlying the right ventricle (V1 and V2). “Rabbit Ears”

slide-55
SLIDE 55

Left Bundle Branch Blocks

What QRS morphology is characteristic? For LBBB the wide QRS complex assumes a characteristic change in shape in those leads

  • pposite the left ventricle (right ventricular

leads - V1 and V2). Broad, deep S waves Normal